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Showing 1 to 12 of 6061 entries
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TUSC7 acts as a tumor suppressor in colorectal cancer.

American journal of translational research

Ren W, Chen S, Liu G, Wang X, Ye H, Xi Y.
PMID: 28979678
Am J Transl Res. 2017 Sep 15;9(9):4026-4035. eCollection 2017.

Increasing studies showed that long non-coding RNAs (lncRNAs) played important roles in the development and progression of tumors. Previous evidences suggested that Tumor suppressor candidate 7 (TUSC7) was involved in several tumors initiation. However, the role of TUSC7 in...

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Ren W, Chen S, Liu G, et al. TUSC7 acts as a tumor suppressor in colorectal cancer. Am J Transl Res. 2017;9(9):4026-4035
Ren, W., Chen, S., Liu, G., Wang, X., Ye, H., & Xi, Y. (2017). TUSC7 acts as a tumor suppressor in colorectal cancer. American journal of translational research, 9(9), 4026-4035.
Ren, Weidan, et al. "TUSC7 acts as a tumor suppressor in colorectal cancer." American journal of translational research vol. 9,9 (2017): 4026-4035.
Ren W, Chen S, Liu G, Wang X, Ye H, Xi Y. TUSC7 acts as a tumor suppressor in colorectal cancer. Am J Transl Res. 2017 Sep 15;9(9):4026-4035. eCollection 2017. PMID: 28979678; PMCID: PMC5622247.
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Fascin-1 is released from proximal tubular cells in response to calcineurin inhibitors (CNIs) and correlates with isometric vacuolization in kidney transplanted patients.

American journal of translational research

Jacobs-Cachá C, Torres IB, López-Hellín J, Cantarell C, Azancot MA, Román A, Moreso F, Serón D, Meseguer A, Sarró E.
PMID: 28979691
Am J Transl Res. 2017 Sep 15;9(9):4173-4183. eCollection 2017.

Immunosuppression based on calcineurin inhibitors (CNIs) has greatly improved organ transplantation, although subsequent nephrotoxicity significantly hinders treatment success. There are no currently available specific soluble biomarkers for CNI-induced nephrotoxicity and diagnosis relies on renal biopsy, which is costly, invasive...

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Jacobs-Cachá C, Torres IB, López-Hellín J, et al. Fascin-1 is released from proximal tubular cells in response to calcineurin inhibitors (CNIs) and correlates with isometric vacuolization in kidney transplanted patients. Am J Transl Res. 2017;9(9):4173-4183
Jacobs-Cachá, C., Torres, I. B., López-Hellín, J., Cantarell, C., Azancot, M. A., Román, A., Moreso, F., Serón, D., Meseguer, A., & Sarró, E. (2017). Fascin-1 is released from proximal tubular cells in response to calcineurin inhibitors (CNIs) and correlates with isometric vacuolization in kidney transplanted patients. American journal of translational research, 9(9), 4173-4183.
Jacobs-Cachá, Conxita, et al. "Fascin-1 is released from proximal tubular cells in response to calcineurin inhibitors (CNIs) and correlates with isometric vacuolization in kidney transplanted patients." American journal of translational research vol. 9,9 (2017): 4173-4183.
Jacobs-Cachá C, Torres IB, López-Hellín J, Cantarell C, Azancot MA, Román A, Moreso F, Serón D, Meseguer A, Sarró E. Fascin-1 is released from proximal tubular cells in response to calcineurin inhibitors (CNIs) and correlates with isometric vacuolization in kidney transplanted patients. Am J Transl Res. 2017 Sep 15;9(9):4173-4183. eCollection 2017. PMID: 28979691; PMCID: PMC5622260.
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MiR-15a-5p negatively regulates cell survival and metastasis by targeting CXCL10 in chronic myeloid leukemia.

American journal of translational research

Chen D, Wu D, Shao K, Ye B, Huang J, Gao Y.
PMID: 28979704
Am J Transl Res. 2017 Sep 15;9(9):4308-4316. eCollection 2017.

Chronic myeloid leukemia (CML) is a common malignant disease from hematopoietic system. Aberrant expression of microRNAs (miRNAs) has been found in CML, however, the roles of many miRNAs including miR-15a-5p in CML are still unknown. In this study, the...

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Chen D, Wu D, Shao K, et al. MiR-15a-5p negatively regulates cell survival and metastasis by targeting CXCL10 in chronic myeloid leukemia. Am J Transl Res. 2017;9(9):4308-4316
Chen, D., Wu, D., Shao, K., Ye, B., Huang, J., & Gao, Y. (2017). MiR-15a-5p negatively regulates cell survival and metastasis by targeting CXCL10 in chronic myeloid leukemia. American journal of translational research, 9(9), 4308-4316.
Chen, Dan, et al. "MiR-15a-5p negatively regulates cell survival and metastasis by targeting CXCL10 in chronic myeloid leukemia." American journal of translational research vol. 9,9 (2017): 4308-4316.
Chen D, Wu D, Shao K, Ye B, Huang J, Gao Y. MiR-15a-5p negatively regulates cell survival and metastasis by targeting CXCL10 in chronic myeloid leukemia. Am J Transl Res. 2017 Sep 15;9(9):4308-4316. eCollection 2017. PMID: 28979704; PMCID: PMC5622273.
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MicroRNA-138 attenuates epithelial-to-mesenchymal transition by targeting SOX4 in clear cell renal cell carcinoma.

American journal of translational research

Liu F, Wu L, Wang A, Xu Y, Luo X, Liu X, Hua Y, Zhang D, Wu S, Lin T, He D, Wei G, Chen S.
PMID: 28861152
Am J Transl Res. 2017 Aug 15;9(8):3611-3622. eCollection 2017.

MicroRNAs (miRNAs) are small, non-coding RNAs which can serve as oncogenes or tumor suppressor genes in human cancers. Herein, the transcriptomic differences of miRNAs in ccRCC were globally assessed using publicly available microarray dataset (GSE71302) from Gene Expression Omnibus...

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Liu F, Wu L, Wang A, et al. MicroRNA-138 attenuates epithelial-to-mesenchymal transition by targeting SOX4 in clear cell renal cell carcinoma. Am J Transl Res. 2017;9(8):3611-3622
Liu, F., Wu, L., Wang, A., Xu, Y., Luo, X., Liu, X., Hua, Y., Zhang, D., Wu, S., Lin, T., He, D., Wei, G., & Chen, S. (2017). MicroRNA-138 attenuates epithelial-to-mesenchymal transition by targeting SOX4 in clear cell renal cell carcinoma. American journal of translational research, 9(8), 3611-3622.
Liu, Feng, et al. "MicroRNA-138 attenuates epithelial-to-mesenchymal transition by targeting SOX4 in clear cell renal cell carcinoma." American journal of translational research vol. 9,8 (2017): 3611-3622.
Liu F, Wu L, Wang A, Xu Y, Luo X, Liu X, Hua Y, Zhang D, Wu S, Lin T, He D, Wei G, Chen S. MicroRNA-138 attenuates epithelial-to-mesenchymal transition by targeting SOX4 in clear cell renal cell carcinoma. Am J Transl Res. 2017 Aug 15;9(8):3611-3622. eCollection 2017. PMID: 28861152; PMCID: PMC5575175.
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Peripheral blood Th9 cells reconstitution and its relationship with acute graft-versus-host disease after matched-sibling peripheral blood hematopoietic stem cell transplantation.

American journal of translational research

Pang N, Xu J, Qu J, Duan X, Yuan H, Chen G, Jiang M, Ding J.
PMID: 28861153
Am J Transl Res. 2017 Aug 15;9(8):3623-3632. eCollection 2017.

T helper type 9 (Th9) cells have recently been identified as a new effector T cell subset. This study is to analyze the reconstitution of Th9 cell after matched sibling peripheral blood hematopoietic stem cell transplantation (MS-PBSCT) and the...

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Pang N, Xu J, Qu J, et al. Peripheral blood Th9 cells reconstitution and its relationship with acute graft-versus-host disease after matched-sibling peripheral blood hematopoietic stem cell transplantation. Am J Transl Res. 2017;9(8):3623-3632
Pang, N., Xu, J., Qu, J., Duan, X., Yuan, H., Chen, G., Jiang, M., & Ding, J. (2017). Peripheral blood Th9 cells reconstitution and its relationship with acute graft-versus-host disease after matched-sibling peripheral blood hematopoietic stem cell transplantation. American journal of translational research, 9(8), 3623-3632.
Pang, Nannan, et al. "Peripheral blood Th9 cells reconstitution and its relationship with acute graft-versus-host disease after matched-sibling peripheral blood hematopoietic stem cell transplantation." American journal of translational research vol. 9,8 (2017): 3623-3632.
Pang N, Xu J, Qu J, Duan X, Yuan H, Chen G, Jiang M, Ding J. Peripheral blood Th9 cells reconstitution and its relationship with acute graft-versus-host disease after matched-sibling peripheral blood hematopoietic stem cell transplantation. Am J Transl Res. 2017 Aug 15;9(8):3623-3632. eCollection 2017. PMID: 28861153; PMCID: PMC5575176.
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Calcitriol promotes survival of experimental random pattern flap via activation of autophagy.

American journal of translational research

Chen L, Zhou K, Chen H, Li S, Lin D, Zhou D.
PMID: 28861155
Am J Transl Res. 2017 Aug 15;9(8):3642-3653. eCollection 2017.

Calcitriol can promote random pattern flap survival and increase autophagy activity. However, effect of autophagy on flap survival after calcitriol treatment, along with the underlying mechanism, is undefined. In this study, the "McFarlane flap" model was established in 72...

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Chen L, Zhou K, Chen H, et al. Calcitriol promotes survival of experimental random pattern flap via activation of autophagy. Am J Transl Res. 2017;9(8):3642-3653
Chen, L., Zhou, K., Chen, H., Li, S., Lin, D., & Zhou, D. (2017). Calcitriol promotes survival of experimental random pattern flap via activation of autophagy. American journal of translational research, 9(8), 3642-3653.
Chen, Long, et al. "Calcitriol promotes survival of experimental random pattern flap via activation of autophagy." American journal of translational research vol. 9,8 (2017): 3642-3653.
Chen L, Zhou K, Chen H, Li S, Lin D, Zhou D. Calcitriol promotes survival of experimental random pattern flap via activation of autophagy. Am J Transl Res. 2017 Aug 15;9(8):3642-3653. eCollection 2017. PMID: 28861155; PMCID: PMC5575178.
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YKL-40 promotes the migration and invasion of prostate cancer cells by regulating epithelial mesenchymal transition.

American journal of translational research

Hao H, Wang L, Chen H, Xie L, Bai T, Liu H, Wang D.
PMID: 28861166
Am J Transl Res. 2017 Aug 15;9(8):3749-3757. eCollection 2017.

OBJECTIVE: This study aims to observe the expression of YKL-40 in prostate cancer and whether YKL-40 can affect the migration and invasion of tumor cells by regulating epithelial mesenchymal transition.METHODS: We collected 14 cases of prostate cancer tissues and...

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Hao H, Wang L, Chen H, et al. YKL-40 promotes the migration and invasion of prostate cancer cells by regulating epithelial mesenchymal transition. Am J Transl Res. 2017;9(8):3749-3757
Hao, H., Wang, L., Chen, H., Xie, L., Bai, T., Liu, H., & Wang, D. (2017). YKL-40 promotes the migration and invasion of prostate cancer cells by regulating epithelial mesenchymal transition. American journal of translational research, 9(8), 3749-3757.
Hao, Hailong, et al. "YKL-40 promotes the migration and invasion of prostate cancer cells by regulating epithelial mesenchymal transition." American journal of translational research vol. 9,8 (2017): 3749-3757.
Hao H, Wang L, Chen H, Xie L, Bai T, Liu H, Wang D. YKL-40 promotes the migration and invasion of prostate cancer cells by regulating epithelial mesenchymal transition. Am J Transl Res. 2017 Aug 15;9(8):3749-3757. eCollection 2017. PMID: 28861166; PMCID: PMC5575189.
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Caragaphenol a induces reactive oxygen species related apoptosis in human gastric cancer cells.

American journal of translational research

Peng QQ, Wang K, Cheng KJ, Yang HG, Qiu JG, Zhang WJ, Jiang QW, Yang Y, Zheng DW, Huang JR, Wei MN, Shi Z, Wang W.
PMID: 28861171
Am J Transl Res. 2017 Aug 15;9(8):3804-3815. eCollection 2017.

Caragaphenol A (CAA) is a novel resveratrol trimer isolated from the roots of Caraganastenophylla. However, the biological activity of CAA is still unknown. In the present study, we investigated the anticancer effects of CAA on gastric cancer cells. CAA...

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Peng QQ, Wang K, Cheng KJ, et al. Caragaphenol a induces reactive oxygen species related apoptosis in human gastric cancer cells. Am J Transl Res. 2017;9(8):3804-3815
Peng, Q. Q., Wang, K., Cheng, K. J., Yang, H. G., Qiu, J. G., Zhang, W. J., Jiang, Q. W., Yang, Y., Zheng, D. W., Huang, J. R., Wei, M. N., Shi, Z., & Wang, W. (2017). Caragaphenol a induces reactive oxygen species related apoptosis in human gastric cancer cells. American journal of translational research, 9(8), 3804-3815.
Peng, Qi-Qi, et al. "Caragaphenol a induces reactive oxygen species related apoptosis in human gastric cancer cells." American journal of translational research vol. 9,8 (2017): 3804-3815.
Peng QQ, Wang K, Cheng KJ, Yang HG, Qiu JG, Zhang WJ, Jiang QW, Yang Y, Zheng DW, Huang JR, Wei MN, Shi Z, Wang W. Caragaphenol a induces reactive oxygen species related apoptosis in human gastric cancer cells. Am J Transl Res. 2017 Aug 15;9(8):3804-3815. eCollection 2017. PMID: 28861171; PMCID: PMC5575194.
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Protective effect of asiatic acid in an experimental cerulein-induced model of acute pancreatitis in mice.

American journal of translational research

Xiao W, Jiang W, Li K, Hu Y, Li S, Zhou L, Wan R.
PMID: 28861174
Am J Transl Res. 2017 Aug 15;9(8):3842-3852. eCollection 2017.

Asiatic acid (AA), a triterpenoid derived from the medicinal plant

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Xiao W, Jiang W, Li K, et al. Protective effect of asiatic acid in an experimental cerulein-induced model of acute pancreatitis in mice. Am J Transl Res. 2017;9(8):3842-3852
Xiao, W., Jiang, W., Li, K., Hu, Y., Li, S., Zhou, L., & Wan, R. (2017). Protective effect of asiatic acid in an experimental cerulein-induced model of acute pancreatitis in mice. American journal of translational research, 9(8), 3842-3852.
Xiao, Wenqin, et al. "Protective effect of asiatic acid in an experimental cerulein-induced model of acute pancreatitis in mice." American journal of translational research vol. 9,8 (2017): 3842-3852.
Xiao W, Jiang W, Li K, Hu Y, Li S, Zhou L, Wan R. Protective effect of asiatic acid in an experimental cerulein-induced model of acute pancreatitis in mice. Am J Transl Res. 2017 Aug 15;9(8):3842-3852. eCollection 2017. PMID: 28861174; PMCID: PMC5575197.
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The STAT3 inhibitor pimozide impedes cell proliferation and induces ROS generation in human osteosarcoma by suppressing catalase expression.

American journal of translational research

Cai N, Zhou W, Ye LL, Chen J, Liang QN, Chang G, Chen JJ.
PMID: 28861175
Am J Transl Res. 2017 Aug 15;9(8):3853-3866. eCollection 2017.

Currently, there is a considerable need to develop new treatments for osteosarcoma (OS), a very aggressive bone cancer. The activation of STAT3 signaling is positively associated with poor prognosis and aggressive progression in OS patients. Our previous study reported...

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Cai N, Zhou W, Ye LL, et al. The STAT3 inhibitor pimozide impedes cell proliferation and induces ROS generation in human osteosarcoma by suppressing catalase expression. Am J Transl Res. 2017;9(8):3853-3866
Cai, N., Zhou, W., Ye, L. L., Chen, J., Liang, Q. N., Chang, G., & Chen, J. J. (2017). The STAT3 inhibitor pimozide impedes cell proliferation and induces ROS generation in human osteosarcoma by suppressing catalase expression. American journal of translational research, 9(8), 3853-3866.
Cai, Nan, et al. "The STAT3 inhibitor pimozide impedes cell proliferation and induces ROS generation in human osteosarcoma by suppressing catalase expression." American journal of translational research vol. 9,8 (2017): 3853-3866.
Cai N, Zhou W, Ye LL, Chen J, Liang QN, Chang G, Chen JJ. The STAT3 inhibitor pimozide impedes cell proliferation and induces ROS generation in human osteosarcoma by suppressing catalase expression. Am J Transl Res. 2017 Aug 15;9(8):3853-3866. eCollection 2017. PMID: 28861175; PMCID: PMC5575198.
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Stachydrine ameliorates pressure overload-induced diastolic heart failure by suppressing myocardial fibrosis.

American journal of translational research

Chen HH, Zhao P, Zhao WX, Tian J, Guo W, Xu M, Zhang C, Lu R.
PMID: 28979698
Am J Transl Res. 2017 Sep 15;9(9):4250-4260. eCollection 2017.

Stachydrine (Sta), a major constituent of

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Chen HH, Zhao P, Zhao WX, et al. Stachydrine ameliorates pressure overload-induced diastolic heart failure by suppressing myocardial fibrosis. Am J Transl Res. 2017;9(9):4250-4260
Chen, H. H., Zhao, P., Zhao, W. X., Tian, J., Guo, W., Xu, M., Zhang, C., & Lu, R. (2017). Stachydrine ameliorates pressure overload-induced diastolic heart failure by suppressing myocardial fibrosis. American journal of translational research, 9(9), 4250-4260.
Chen, Hui-Hua, et al. "Stachydrine ameliorates pressure overload-induced diastolic heart failure by suppressing myocardial fibrosis." American journal of translational research vol. 9,9 (2017): 4250-4260.
Chen HH, Zhao P, Zhao WX, Tian J, Guo W, Xu M, Zhang C, Lu R. Stachydrine ameliorates pressure overload-induced diastolic heart failure by suppressing myocardial fibrosis. Am J Transl Res. 2017 Sep 15;9(9):4250-4260. eCollection 2017. PMID: 28979698; PMCID: PMC5622267.
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The negative regulation of miR-149-5p in melanoma cell survival and apoptosis by targeting LRIG2.

American journal of translational research

Chen W, Zhang J, Xu H, Dai J, Zhang X.
PMID: 28979706
Am J Transl Res. 2017 Sep 15;9(9):4331-4340. eCollection 2017.

MicroRNAs (miRNAs) are key regulators of diverse biological processes in tumor progression including melanoma. LRIG2 is reported as an oncogene in cancer, however, little is known regarding the molecular and functions of LRIG2 in melanoma. In this study, we...

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Chen W, Zhang J, Xu H, et al. The negative regulation of miR-149-5p in melanoma cell survival and apoptosis by targeting LRIG2. Am J Transl Res. 2017;9(9):4331-4340
Chen, W., Zhang, J., Xu, H., Dai, J., & Zhang, X. (2017). The negative regulation of miR-149-5p in melanoma cell survival and apoptosis by targeting LRIG2. American journal of translational research, 9(9), 4331-4340.
Chen, Wenqi, et al. "The negative regulation of miR-149-5p in melanoma cell survival and apoptosis by targeting LRIG2." American journal of translational research vol. 9,9 (2017): 4331-4340.
Chen W, Zhang J, Xu H, Dai J, Zhang X. The negative regulation of miR-149-5p in melanoma cell survival and apoptosis by targeting LRIG2. Am J Transl Res. 2017 Sep 15;9(9):4331-4340. eCollection 2017. PMID: 28979706; PMCID: PMC5622275.
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Showing 1 to 12 of 6061 entries